Saturday, March 26, 2016

Neonatal jaundice

It will affect the intelligence of neonatal jaundice

Neonatal physiological jaundice neonatal period is characteristic of a normal phenomenon, because the fetus in utero hypoxia, erythropoietin blood too, and the kind of immature red blood cells more susceptible to damage, birth after causing too much bilirubin generated about twice that of adults; on the other hand, immature neonatal liver function, restricted metabolism of bilirubin and other factors, neonatal jaundice phenomenon for some time. Physiological jaundice usually 2 to 3 days after the child was born full-term, peaked at 4-5 days to 5-7 days subsided.

Performance of children with skin, eye and oral mucosa white yellow, from mild to severe. Usually on the face and chest obvious, but the palms and soles of the feet are not yellow. 4 to 6 days the most obvious, full-term children at 10 to 14 days after birth subsided, sustainable preterm children to the first three weeks.

During this period, children generally in good condition, no other discomfort performance. Although neonatal physiological jaundice is a normal phenomenon, but parents should also pay attention closely observed. In general, the physiological jaundice jaundice lighter, lower serum bilirubin concentration, it does not affect intelligence in children.

How to treat jaundice

Light therapy

No reduction of serum bilirubin is a simple and effective method for binding. The neonatal phototherapy Woyu box, with black goggles to protect his eyes, so as not to damage the retina, the perineum, anal covered with diapers, the rest are bare. Single-sided or double-sided light irradiation for 2 to 48 hours (usually not more than 4 days), bilirubin to 7 mg / dl or less stop treatment.

Blood transfusion therapy


Exchange transfusion can effectively reduce bilirubin, swapped out the sensitized red blood cells and reduce anemia. But shake requires certain conditions, can produce some adverse reactions, it should be strictly controlled indications.

medical treatement

Supply albumin, correction of metabolic acidosis, liver enzyme inducers (such as phenobarbital), intravenous immune globulin.

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