TCM treatment of male infertility caused approach

In Chinese medicine dialectical therapy on the basis of the following selection of proprietary Chinese medicines: (1) is a type of kidney deficiency, Bushen to fill skilled Arresting Jing Guan, taking Liuweidihuang balls, five of the yellow pills, Wuzi Yanzong decade. (2) is a Liver-Qi stagnancy, to Shugan Jieyu, the choice of Xiaoyao pill, Shugan pill, in the Shaogan LiuJunZi decade. (3) the type of traveler Mingmen, kidney-YANG Yi Zhuang, the choice Yougui pills, tablets, Bushen fit. (4) is a type of kidney deficiency, Bushen Yi Yin, the choice Zhibai Dihuang Wan, Jiawei Huang decade. (5) is a type of blood, to Blood Circulation, the choice of lower Zhuyu pills, Brewers decade. (6) is a type of phlegm damp to be filled, preferably removing expectorant, Spleen and stomach, the choice of Xiangsha LiuJunZi pills, Jiawei Xiangsha Zhizhu decade.

Prevention and treatment of secondary infertility

A. intensify gender premarital health education, college students and young couples should seriously re-education or sex education. B. The inflammatory infection, disinfection work closely, and medical workers must first strictly enforced. Second, in order to avoid surgery, tissue trauma and postoperative patients should be carefully instructed attention. As in patients with postoperative hemorrhage or acute inflammation encountered such circumstances, it must better conditions to the nearest hospital emergency department. Not worried about privacy exposed delays conditions had deteriorated to a pelvic abscess Swelling, and a long time to chronic pelvic inflammatory disease caused infertility, or even life-threatening. C. clinic patients are chronically ill. In its treatment, the first need to identify disease, menstrual disorders, ovulation dysfunction, or obstructed tubal patency distal water, adhesion, whether chronic pelvic inflammatory disease, and mass. Uterine location flexor serious, severe cervical secretion of excessive erosion can affect pregnancy. Clinically, for the treatment or classification given by the Chinese theory of temperature and Qualcomm, reduced premium, or the right to adjust the overall combination of traditional Chinese and western medicine treatment can be received more satisfied with the effect.

Infertility classification

Infertility can be divided into: (1) primary and secondary infertility infertility, that the former marriage has never been pregnant, which means that there had been pregnancy, but nearly two years no longer pregnancy; (2) relative and absolute sterility, infertility refers to the former couple have one anatomical or functional defects can not be corrected and not pregnant, which means one of a couple of factors, beyond the proper treatment may become pregnant; (3) male infertility or female infertility, according to infertility factor in the man or the woman and distinction; (4) organic sterile and functional infertility, the former refers to the organic genital lesions caused by infertility, which refers to the main endocrine abnormalities caused by infertility.

What is chronic lymphocytic leukemia symptoms

First, the general symptoms: onset implicit attack, the slow progress of the tumor itself can cause fatigue, weakness, weight loss and other symptoms. Itching skin, pigmentation, erythema, exfoliative dermatitis and herpes zoster. Second, infections: Because of immune abnormalities in immune dysfunction and various infections, the most common respiratory infections, skin, gastrointestinal tract, urinary tract, such as sepsis. Third, liver and spleen lymphadenopathy: superficial lymphadenopathy slow leaching is the most common signs, with the condition progress from small to large and small increase, from local to the whole body. IV, skin damage.

The treatment of acute leukemia in children principle

The chemotherapy of acute leukemia in children is based on the principles of options, as far as possible, adopt a strong induction chemotherapy programs, joint use, adequate, intermittent, alternately, long-term treatment approach. Leukemia treatment of the fundamental purpose is to enable long-term disease-free survival of patients until cured. Therefore, to be followed in the treatment of acute leukemia classification by type of program to use the principle of selection of the best current chemotherapy program at home and abroad to improve the quality of complete remission, and promptly formal effective complete remission after treatment of leukemia and central nervous system prevention and treatment, we will strive for the greatest possible number of patients cured. (1) combination: synergies can reduce relapse, remission and long-term survival rates improve. (2) adequate administration: In induction phase of the consolidation of the drug enough to kill the greatest number of leukemia cells, bone marrow suppression quickly reached state in the future to reduce the recurrence is extremely important. (3) the type administration: different types of leukemia in the treatment programs are different, they should be of Treatment. (4) intermittent chemotherapy: Intermittent between the two treatment facilitate the resumption of normal hematopoietic cells, and in the telogen entered the proliferation of leukemia cells cycle of chemotherapy in the next easily be killed. (5) long-term treatment: prevent relapse and improve long-term survival rates. For several years in the chemotherapy and chemotherapy can be summarized into two basic programs: ① remission induction therapy (including differentiation therapy); ② remission after treatment (including the consolidation treatment, intensive therapy, and maintenance therapy in the prevention of extramedullary leukemia).

What are the characteristics of the elderly leukemia

The elderly in hematopoietic malignancies, chronic lymphocytic leukemia, lymphoma, multiple myeloma is common, myeloproliferative diseases include bone marrow fibrosis (MDS), really RBC hyperplasia, also of primary thrombocythemia for common to the elderly. Elderly relatively high incidence of leukemia. Acute leukemia to acute monocytic leukemia the most common, chronic leukemia to the most common chronic lymphocytic leukemia. Elderly adults with acute leukemia compared to relatively slow onset, and conceal, onset time of the main symptoms of anemia and other symptoms relatively rare. Some of the elderly are manifested as low proliferative leukemia, reduce blood cells, bone marrow hyperplasia lower. Because the elderly poor physique, often associated with other diseases, not the strong chemotherapy tolerance, poor prognosis. About two-thirds of slow leaching for in people older than 60. Onset slow, often no early symptoms, physical examination or inquiry in more blood was found. Clinical to lymphadenopathy and hepatosplenomegaly as the main performance. Due to the slow progress in the course, the majority of patients with longer survival time. Hairy cell leukemia is a relatively rare, relatively slow leukemia, the majority of patients for the elderly. Misprision of disease onset, patients often due to fatigue, giant spleen were found. Blood as a whole blood cell reduction. The slow progress of this disease, patients with longer survival time.

How to distinguish between acute and chronic leukemia

The distinction between acute and chronic leukemia incidence in addition to the, but also mainly based on: (1) life expectancy or natural process untreated patients with acute leukemia average life expectancy of less than one year, and chronic leukemia average life expectancy for 1 - 3 years; (2) leukemia cell differentiation, the maturity of the acute leukemia bone marrow or peripheral blood cells in the main for the original leukemia (lymphatic system, or granulocyte monocyte line) cells, and chronic leukemia in the bone marrow and peripheral blood main the mature phase of the tablets and naive cells (chronic myeloid leukemia) or mature small lymphocytes (chronic lymphocytic leukemia).

Chronic lymphocytic leukemia diagnosis standards

(1), history and symptoms ⑴ history question: slow onset, patients have no obvious symptoms, history-taking should pay attention to whether there is a fever, night sweats, vulnerable performance. ⑵ clinical symptoms: fatigue, weight loss, lack of appetite, night sweats, physical decline, fever, sometimes skin itching. (2), physical examination found Systemic lymphadenopathy, quality Medium hard, movable, mutual adhesion advanced integration; mildly enlarged liver and spleen, advanced significantly enlarged spleen. Bone pain not obvious. Advanced skin visible bleeding point. (C), auxiliary inspection 1. Blood: are cell anemia is pigment. WBC count> 10 × 109 / L, classification: lymphocyte> 50%, the absolute value> 5.0 × 109 / L; mature lymphocyte predominance, we can see that naive lymphocytes and different types of lymphocytes. Platelet normal or reduced. 2. Bone marrow as: hypertrophic extremely active to active, mature lymphocyte proliferation obviously, accounting for more than 40 percent of the original, naive lymphocytes <10%.>

Primary peritonitis

Outlined Primary peritonitis pulp cavity is not the original source of morbidity, Pathogens through blood supply, lymphatic, or intestinal wall channels such as female reproductive tract caused by invasive abdominal peritonitis. Most of the patients systemic poor, the girl child and adult chronic nephritis, or cirrhosis with ascites high incidence of patients; pathogens mostly hemolytic streptococcus, Streptococcus pneumoniae and Escherichia coli. The main symptoms of the disease is a sudden onset of acute abdominal pain starting position not clear, quickly spread to the entire abdomen, diagnosis of this disease is the key to exclude secondary peritonitis.
Clinical manifestations 1. Often prior to the onset of upper respiratory tract infection or kidney disease, scarlet fever, liver cirrhosis and ascites occurred when the lower immune function; 2. Main symptoms are sudden onset of acute abdominal pain, starting position is not clear, very quickly diffuse to the whole abdominal 3. associated with nausea and vomiting, fever, systemic poisoning symptoms fast pulse; 4. abdominal distention, abdominal tension-wide, tenderness anti Music weaken or disappear.
Diagnosis 1. Persistent abdominal pain, starting position is not clear, the whole diffuse abdominal soon. 2. Obviously peritoneal irritation sign,sound disappear. 3. Drawn pus abdominal wear thin without the smell, in addition to films that examination Gram-positive cocci.

Treatment of primary peritonitis

Treatment principles 1. Defined as primary peritonitis, Non-surgical treatment available, including the choice of Streptococcus infections sensitive or broad-spectrum antibiotics to control infection; infusion therapy and support. 2. Such as non-surgical therapy is not effective, illness gradually worsened or secondary peritonitis can not be ruled out, should laparotomy.
Medication principle 1. Early mild cases with penicillin G, ampicillin, metronidazole, erythromycin anti-infection treatment observation. 2. Peritonitis after surgery to recovery or to complications. Serious complications can choose ceftriaxone human albumin drug treatment.
Evaluation 1. Cured: Clinical symptoms and signs disappeared. 2. Turn for the better: Clinical symptoms and signs improved. 3. Surprised me: Clinical symptoms and signs of improvement.

Kawasaki disease hazard to children

Kawasaki disease is a vasculitis syndrome, Also known as mucosal skin lymph node node syndrome, occurs in 2-4 years old children, common to boys. In recent years, the incidence rate has increased. Kawasaki disease is the greatest damage against coronary artery, coronary artery lesions in children is the main reason, but also adult after the potential risk factors for coronary heart disease. In China in July-September incidence more, therefore, at the turn of the summer and fall, the parents should be particularly vigilant in Kawasaki disease in infants. Children in general are more than before on the incidence of respiratory infection prodromal symptoms. Sustained high fever and no rebate 1-2 weeks, the eyes conjunctival hyperemia, your lips lesion hemorrhage, congestive oral mucosa, and tongue-like surface seems Yangmei, a systemic form of diversification rash, edema flushing and hand-foot-rigid cervical lymphadenopathy. These symptoms persisted, in the fingers and toes along the terminal at the junction of nail and skin characteristic of a membrane-like peeling, can also be found around the anus peeling. Some children with Kawasaki disease can be complicated by heart, brain, liver, lung, kidney and other organ damage, of which the most prominent cardiovascular damage, most occurred in incidence after 2-3 weeks, sustainable few months to several years. Kawasaki disease complicated by the type of cardiovascular damage, including coronary artery aneurysms, coronary expansion, such as coronary artery stenosis or occlusion. Aneurysms can be single or multiple, mostly dissipated to be 1-2 years, but partial wall fibrosis can be prompted intimal hyperplasia, leading to coronary not effective expansion, can be a serious thrombosis, luminal stenosis and occlusion myocardial infarction , can lead to serious cardiac ischemic symptoms or develop into ischemic heart disease, died of Kawasaki disease is the main reason. Kawasaki disease hazards so large, so when the children fever with rash, mucosal damage and enlarged lymph nodes, and other symptoms, parents should be timely children to go to the hospital treatment. Because the reasons for the cause of Kawasaki disease is not very clear, it is still not a good prevention measures its incidence, but in the majority of children have had prior to the onset of upper respiratory tract infection, therefore, at the turn of the summer and fall, should be avoided as far as possible upper respiratory infection. Regardless of whether children with Kawasaki disease coronary artery lesions, should pay attention to diet is reasonable, as far as possible avoid the occurrence of hyperlipidemia and hypertension, to reduce the occurrence of coronary heart disease when they grow up and dangerous. In addition, children with Kawasaki disease too, again incidence rate of 1% -3%, it can not be taken lightly. Less attention usually came down with a cold should avoid recurrence.

Henoch - Schonlein Purpura the differential diagnosis

First, idiopathic thrombocytopenic purpura according to the shape of skin purpura is not above the skin, Distribution of asymmetry and platelet count decrease, it is not difficult to identify. Henoch - Schonlein purpura with rashes such as vascular nerve edema, erythema shape urticaria or more distinction. Second, sepsis meningococcal septicemia caused by rash and Purpura similar, but the symptoms of poisoning weight, interleukin significantly increased, the shattered rash seized smear positive for bacteria. Third, the festival can be both rheumatoid arthritis pain and fever, a former Purpura more difficult to identify, with the development of disease, skin purpura, help identification. 4. Intestinal Invagination was particularly prevalent in infants. If children, abdominal touched mass, muscular tension of this disease should be suspected. Perspective barium enema may be differential. But anaphylactoid purpura can be accompanied by intestinal sets again and again, it should be much attention. 5, appendicitis can be both a and right lower abdominal pain with tenderness. But not anaphylactoid purpura abdominal tension, skin purpura there may be differential.

The method of diagnosis of urinary tract infection What?

Urinary tract infections, as the inspection to be done with the specific circumstances of the case. Urine routine inspection is the clinical application of the most extensive and most user-friendly method, Although it has not confirmed the value of urinary tract infection, but the combination of clinical symptoms, clinicians often can be used as a preliminary basis for the diagnosis of urinary tract infection. Aidi count is one of the ways urine microscopy can be used as the auxiliary urine routine inspection. Urine culture is a diagnosis of urinary tract infection means of inspection, only urine culture can be positive diagnosis of urinary tract infection, or the diagnosis can not be established. Urine culture and drug-sensitive test for the selection of antibiotic treatment of important reference value.
On the other suspect with the disease or suspected urinary tract infection patients do kidney, bladder ultrasound, X-ray inspection (including abdominal plain film, IVP), and even CT examination, except to renal tuberculosis, urinary tract stones, kidney plot water, renal cysts, kidney tumors, such as urinary tract malformations.
Uncertainty is the upper urinary tract infection, or patients with lower urinary tract infection, urine checks parcels in the cell antibodies (ACB), urinary lysozyme, β2 - microglobulin, and so have some clinical significance. Recurrent urinary tract infection, Touching should regularly check kidney function.

Urinary tract infection in children

Urinary tract infection by the bacteria directly caused by invasive urinary tract inflammation, Including urethritis, cystitis, pyelonephritis, and so on. Because breast cancer in children after difficult position, collectively called urinary tract infection. There are symptoms parents should consider children suffering from urinary tract infection? The adult child may have similar symptoms, such as urgency, frequency, urinary pain. While infants are often fever, crying, quite disturbed, anorexia, wet his pants as the main symptoms. The frequency of small babies, often soaked diaper, prone diaper dermatitis, perineal skin flushing.
Home care points 1. Children suffering from urinary tract infection if fever needs bed rest. Systemic all timely, appropriate activities. 2. More drinking water. Infants should Qin, older children can be encouraged to play a sick child more water rinse urethral role. 3. Diet. Suitable to light, eat more fruits and vegetables. 4. Maintain cleanliness of genitalia. Infants should diapers, feces after cleaning the hip perineal Nitrofurazone hip bath can be used if ulcer after Tu tetracycline cortisone ointment. 5. Urinary tract infection drug resistance should be taken after meals, such as bark feeding Dante, in order to reduce gastrointestinal reactions. 6. Observation urine color, urine output and urinary frequency changes. There are nausea, vomiting, and other drugs to doctors reaction reflected.
Note 1. For urinary tract infection first do a good job of prevention, such as to give their children every night, especially girls cleansing genital, have their own pots, towels, and abandoning shared with adults. Infants try not to wear pants, regular replacement of underwear, underpants. 2. After the urine pain medication, the symptoms of urgency faster will turn for the better, gradually normal urine tests, at this time the most important thing is to continue ophthalmic drugs by doctors, patients must not see little children on their own withdrawal symptoms. Generally speaking of drugs two weeks, even those with severe extension. Two weeks after stopping to review urine, every three to six months review time, no relapse for two years to recover. 3. Acute urinary tract infection if not reasonable treatment, or lack of medication treatment, urinary tract infection deferred, course to more than six months or more to hair, known as chronic urinary tract infection, for the performance of recurring symptoms of acute exacerbation , and ultimately affect renal function, and may even cause uremia.

Urinary tract infection caused abnormal urine

Urinary tract infection can cause abnormal changes in the urine, A common bacteria urine, pyuria, such as hematuria and gas urine.
(1) bacterial urinary bacteria in the urine is urine bacteria. Most women and vaginal vestibule of the urethra mouth 1 to 2 cm from the rectum of potential pathogens, voiding, the bacteria can be mixed in the urine; sexual intercourse or catheterization, will be easy within the urethra into the bladder bacteria, cause infection. Non-specific infection pathogens 70% ~ 80% for the Gram-negative bacteria, including Escherichia coli, Proteus, the Deputy Escherichia coli, Pseudomonas aeruginosa and gas production bacilli. The remaining 20% of pathogens for Gram-positive cocci infections, including staphylococcus, streptococcus, and so on.
(2) pyuria pyuria that urine in. Common causes of a specific infection and non-specific infection two categories. Non-specific infection including pyelonephritis and renal empyema, around abscess rupture with the same kidney, bladder inflammation, urethritis and Annex organ inflammation (such as pelvic abscess, prostatitis or abscess). A common bacteria Escherichia coli, Proteus, such as Staphylococcus aureus. Other infections, such as mycoplasma, fungi, trichomonas, herpes virus can also be seen. Specific mainly tuberculosis infection with gonorrhea. In addition, urinary tract neoplasms, stones, injury, benign prostatic hyperplasia, neurogenic bladder, urethral stricture and urinary tract anomalies such as congenital or secondary infection causing obstruction can be caused pyuria.
(3) the reasons for hematuria hematuria about 40 species, most of the urinary system caused by the disease itself, and a few other systems with systemic diseases. According to a statistics, the incidence of hematuria common cause order: urinary tract infections, stones, tumors, benign prostatic hyperplasia, injury, tuberculosis, and so on. Urinary tract infection caused by hematuria, the most common. Hematuria with bladder disease that stimulate more symptoms of urinary tract next to the most acute cystitis see. Hematuria performance for the end, even for the whole of hematuria, with frequency, urgency. If timely treatment, after a few days can ease the symptoms. If patients with high fever, chills, flank pain, and other symptoms, should consider acute pyelonephritis. Chronic cystitis prolonged or repeated illness is acute, the merger are rare. Acute prostatitis hematuria will have terminals, in addition to stimulating the bladder with symptoms, systemic symptoms such as high fever, chills, nausea and vomiting, weakness, and so is very obvious. Seminal Vesiculitis in the acute phase and acute prostatitis similar, abdominal pain, the need to differentiate with other acute abdomen. Young patients stimulate bladder symptoms and terminals hematuria, the longer course, the general antibiotic treatment fails, should consider renal tuberculosis, need further checks to clear diagnosis. Bladder cancer patients with tumors larger if, in particular tumor deep myometrial invasion, but also stimulate the bladder symptoms appeared that the course has entered late. In addition, the anti-cancer drug cyclophosphamide intravesical, can cause hemorrhagic cystitis; cervical cancer or bladder cancer after radiation therapy, radiation cystitis can cause, but also of similar symptoms.
(4) Gas Gas urinary voiding urine that is emerging in urine when gas from emphysematous cystitis or caused by gas pyelonephritis. Emphysematous cystitis is a bladder wall cavity or an inflammation of gas exist in the incidence of diabetes in patients with high. For pathogens Escherichia coli, Bacillus gas production (including Proteus), Staphylococcus aureus, Streptococcus and yeast, the gastrointestinal tract, lungs or skin for primary tumors, blood or injury Department urothelial lesions into the urinary tract location, glucose glycolysis and produced carbon dioxide. Non-diabetic patients with long-term glucose injection or the indwelling catheter, cause injury and bladder infection, to multiplication of bacteria. Gas pyelonephritis for outbreaks of common kidney infection, kidney and renal around both gas. Often with sepsis, kidney empyema and necrosis. But on the more common clinical gas urine from the urinary tract is between the intestine and the formation of fistulas. Except caused injury or surgery, except more fistula due to pathologic tumor, tuberculosis, Crohn result. Apart from the gas in urine, sometimes from fecal residue, food debris, tumor size, cheese - like substances.

The causes for the cause hemorrhoids

Hemorrhoids anorectal Department is due to blood circulation sluggish, Venous blood returning obstacles caused buckling expansion vein formation of soft Mission. The causes hemorrhoids caused many, and is generally believed that the following factors: Local factors vascular anatomy less elastic fibers, rectal veins and lack of venous valve, dilated blood vessels easily buckling stasis, a hemorrhoid. Long Li sedentary upright posture, anal lower position by the force of gravity, the Department of rectal blood returning impeded, easy congestive into hemorrhoids. Local factors anal infection or intestinal inflammation, caused inflammation of tissues around the lower rectum, vascular embrittlement, congestive secondary vascular dilation caused or exacerbated hemorrhoids. Constipation and abnormal defecation intractable constipation, defecation long time, defecation, hard earned or long-term diarrhea, abdominal pressure will increase, the Ministry of congestive anorectal varicose veins, expanding about anal muscle and muscle relaxation Rectal prolapse prone separation hemorrhoids. Pregnancy and childbirth pregnancy increases abdominal pressure, congestive pelvic vein, the lower part of the rectum when congestion forced labor, straight from the gut. Genetic factors have reported about 44% of patients have family history of hemorrhoids. Dietary factors like to bitter, alcoholic drinks and tobacco hyperstimulation anorectal. High frail, long illness anal sphincter relaxation weakness, caused or exacerbated hemorrhoids prolapse. Portal hypertension cirrhosis and portal vein thrombosis portal hypertension caused inflammation, hemorrhoids caused increased pressure within the venous plexus, buckling expansion. Exertion over long-term weight-bearing travel, Chronic Cough Jiushu,and other long-term abdominal pressure increases, rectal veins expansion straight from the gut.

The treatment of acute cholecystitis What are the principles

For patients with acute cholecystitis, The general non-surgical treatment, symptoms can be alleviated and more, after re-elective surgery.
Non-surgical therapy, including bed rest, fasting, transfusion to correct water and electrolyte imbalance, vitamin and antibiotics, if necessary, to gastrointestinal decompression. Abdominal pain can be given when Antispasmodic agents and analgesic agents such as atropine, meperidine, at the same time should be closely observing the condition changes. Any of the following circumstances, it shall be the short term symptomatic treatment for, the purposes of emergency surgery:
(1) clinical symptoms heavy and difficult to mitigate, enlarged gallbladder, and the tension are more likely to have perforation.
(2) abdominal tenderness obviously, muscular stiffness, peritoneal irritation symptoms, or to observe the course of treatment, signs add to the abdomen.
(3) suppurative cholecystitis have chills, high fever, were significantly higher leukocyte.
(4) general acute cholecystitis in the non-surgical treatment failed to alleviate the symptoms or worsening of the illness are.
(5) elderly patients susceptible to gangrene and perforation of the gallbladder, the symptoms early surgery should be heavier.

What are the performance urticaria

Urticaria commonly known as "rubella block," "Dan the Wind" is a common multiple. Urticaria most prominent manifestation of this is suddenly itching skin, Then in the flat above the skin Wind Mission, and some redness, some Blushing, from the more different size and shape, but clearly the border, accompanied by itching, night heavier. Attack time uncertain. Wind Mission gradually spread, suspicious integration into films, both confined to a particular location, but also systemic generalized. A small number of patients can with nausea, vomiting, accelerated heartbeat, headache, diarrhea and, sometimes often fever. According to the course length can be divided into acute and chronic urticaria two types. The general incidence of acute urticaria sharp, the majority can find a cause, food and children to secondary infections common, particularly for upper respiratory tract infection, purulent tonsils and gastro-intestinal infections. In one week around the course. Most of the patients can be dissipated in a few days. Chronic urticaria, duration greater than 6-12 weeks, the wind Mission repeated attack, 80% -90% of the patients children often can not find cause to the treatment difficult.
Combat is the urticaria, urticaria suspicious Although many and complex causes, but carefully identify trigger factors, banned sensitized suspicious banned drugs and short-term food, active infection control and treatment is the key. This was followed by the Oral anti-allergic drugs, 1 - 2-year-old oral cents drops, such as 2-year-old following oral chlorpheniramine, 2-6-year-old oral the special non-small granules, preschool and school-age children oral opened. Appropriate vitamin C, calcium helps rash dissipated. If the merger bacterial infection, antibiotics should be given active, infection is available Austrian Fortune, ribavirin, acyclovir, can cure fungal infections such as mycophenolate SHU. If parasitic infection of the need for insecticide, chronic urticaria often needed two kinds above. Serious urticaria take to the hospital, if necessary, oxygen, intravenous transfusion hormones, boost medicines. Oral medicine can be properly chosen to drive wind Jianpi, Liangxue detoxification. The medication used calamine lotion antipruritic.

What is the blood measles urticaria?

Blood measles urticaria in children also called lichen planus, Winter and early autumn is the most common form of pediatric allergic skin diseases. Most infants and pre-school children is high. Generally considered the incidence of insect bites is the direct incentives, the allergic qualities body is foundation. Insect common pathogenic mosquito, Moskit, fleas, bedbugs, mites, Ceratopogonidae, ticks, etc., because of specific pathogens of insects, from place to place, because the seasons are different, and some very small insects, the naked eye can not see. When children in the grass,or under the carpet play or contact cats, dogs and other animals when unconsciously insect bites, insect saliva into the skin of children, cause allergic reactions, blood measles urticaria occurred, the body more sensitive, the more severe the rash also, some people think with certain foods such as fish and shrimp allergies or digestive dysfunction related. This disease of the skin damage generally has three forms, one for the spindle,shape soybeans large rubella peanuts big red block-like edema of blood measles,after the rash can be increased more, some are pseudo-foot , the center that a pile of small blisters, lentils for another big nail to build large, thick-walled shiny tension large blisters, another is a big red papules adzuki bean, hard substrate, the shiny top of a small blisters. This much like chickenpox rash, and some people did not know, and it often confused with chickenpox, varicella often fever, oral mucosa damage can also occur, more often facial rash, Papular urticaria often occur in the limbs, trunk the neck or hip or irregular groups scattered distribution, normally have obviously itching, night especially important. After proper treatment, most can be cured about a week, but often recurrent, sometimes future can be a secondary bacterial infection impetigo, or to cause great pain in children. How to Combat Papular urticaria? We must prevent and Papular urticaria must first pay attention to the eradication of harmful insects, bedding, furniture, carpets, corner etc. should regularly sprayed Ling, and other pesticides. To eliminate the dogs, cats and birds kept in the home or other animals of fleas, mites and other parasites. Meanwhile, we should pay attention to maintaining skin hygiene, eat more vegetables, fruits, gastrointestinal dysfunction should prompt medical treatment. The incidence may have been taken such as chlorpheniramine and diphenhydramine, also can take proprietary Chinese medicines horns of poison pill,payment of poison powder, pediatric-orange, can be treated with local white lotion or water only itch agent. If secondary infection or chronic should be timely to hospitals for treatment.

Clinical manifestations of congenital ichthyosis

(1) autosomal dominant hereditary unusual ichthyosis,This is a common type of light ichthyosis, substituting one or both parents were home sick more often with catalyst, but no gender differences. Often, a child morbidity, and increased with age, to the most obvious symptoms of adolescence, that is, after the cessation of development. Lesions performance of different degrees of seriousness, the only light to the winter dry skin, no obvious,after powder loading debris, known as dry skin disease. I see hair follicle or in the size of a tip of the hair follicle and brown, without consciously disease, known as hair follicles of ichthyosis (ichthyos; sfoll; cularis). In addition to common skin drying, the idea that or deep brown, central fixation, the free edge. Also was gray and white, with mica-like sheen, gloss of ichthyosis called. More significantly abnormal proliferation of keratinocytes; lcm above can be thick, rough surface, or a spine-like uplift papillary surface dark gray, like bark, herein referred to as superb ichthyosis.
Friends of the disease in the symmetric stretch limbs and trunk side, particularly Zhouxi side for a stretch. Severe cases can be spread to the body, with palmoplantar hyperkeratosis, and that the saddle cleft (toe) A change. Hill like face, scalp, elbow Waterloo, armpit, gills Waterloo, genital, and Tungou often not be violated, or only mildly. Usually without conscious of the winter due to decreased secretion of sweat glands and sebaceous gland, skin drying, and consciously itching discomfort, if the lesion is flu pain. Often patients with atopic constitution, such as fever and asthma subtilis.
Pathology: cuticle significant thickening, particle layer thinning or disappeared. The stratum corneum and stratum corneum cells thickening the desmosome slow degradation, cuticle off the normal cells failed. Spike thin layer cell, basal layer pigment increase. As Hyperkeratosis enter into hair follicle formation of hair follicles Kok suppository. Adenovirus can sweat gland cortical atrophy. No significant changes in the dermis, or in a small number of perivascular lymphocytic infiltration.
(2) of the unusual fish genetic disease: less see. As the disease gene in the X chromosome, is found only in males, may infant after birth or morbidity. Lesions on the type and slightly different large and significant, a tawny or sewage black large, dry rough skin, often around the body, axillary, and the elbow of Waterloo Waterloo, and so can the Department involvement; back especially important than the abdomen . If facial involvement is limited to the ears before and facial aspect. Hair follicle keratosis not normally occur. Palmoplantar from normal skin, skin lesions did not increase with age and reduce, and sometimes it by drama. Female carriers of the disease, does not generally incidence, sometimes only found in the forearm and leg appeared thin. After flexibility and posterior corneal layer membrane turbidity can be a small point, and do not affect vision.
Pathological Hyperkeratosis, normal or granular layer thickness, spine mild thickening layer, the dermis no significant changes can sometimes see in a perivascular lymphocytic infiltration.
Usually seen in clinical samples congenital ichthyosis Erythroderma disease and lamellar ichthyosis as follows:
(C) epidermolysis bullosa of ichthyosis Hyperkeratosis bullous disease, also known as congenital ichthyosis kind Erythroderma disease, with high distortion rate for the autosomal dominant genetic disease. Clinical rare. After birth or after birth a few months, there are generalized and limitations of damage. Pan-onset systemic birth when there is armor-like thick Health after falling out of the emerging pan-sexual flushing, stripping showed moist surface, erythema may gradually disappear, thick wart can happen again -. Only those limitations in the limbs and wrinkle flexor side of the wall will have thick horny tablets. Weight may have defects - like warts hyperplasia or hyperplasia was damage-limitation ask that porcupine-like ichthyosis. The disease started a few years, there will be the small wooden ranging from relaxation of water scars or scars. The use of antibiotics to control infection, and reduce the occurrence of water scars. Palm involvement can affect function.
Pathological epidermal Hyperkeratosis, granular cell layer thickness, the spine of the upper reticular cell vacuolization, only sometimes desmosome associated with the side of spine cell lysis spinous process of epidermal cells, epidermal water scars visible within or scars, a chronic inflammatory dermal shallow infiltration.
(D) lamellar ichthyosis of autosomal recessive genetic, which is a whole body after birth extensive cotton rubbery membrane were tightly wrapped, multi-valgus cause eyelids and lips. A few days later the membrane off, a broad indemnity chronic skin flushing, there are gray or polygonal or rhomboid large, central fixation, the free edge. Often symmetry of the body to physically flexor side, elbow Waterloo, the Waterloo, armpit and genital and other units more distinctly. Palmoplantar hyperkeratosis, fingernails and excessive hair growth. After slow course, there could be life to adulthood erythroderma ease, but still exist. The disease is autosomal recessive inheritance.
Pathology: epidermal middle of Hyperkeratosis, focal keratosis incomplete, granular layer and part of spike cell layer thickening, epidermal sudden extension, a chronic inflammatory dermal shallow infiltration.
2. Treatment:
Yang internal medicine doctor with pure, clean and treatment of ichthyosis, follicular keratosis of, a good effect, the drug from Chinese medicine Astragalus 12 - 18 g. Polygonatum 15 - 18 g. system Shouwu 15 - 30 g. dog Qi 12 -- 18 g. black sesame 12 - 18 g. snake retreat 10 - 15 g. Ling cents 10 - 15 g. Health steroid 18 - 30 g. ginseng 15 - 24 g, and other drug component. skin with moisture. active. flooding Wind the antipruritic effect. with other rub, better efficacy.

Is seborrheic keratosis senile plaques

Also known as seborrheic keratosis elderly wart disease, senile plaques and basal cell papilloma, A benign skin tumors. Most occur in the elderly, although young people can be seen, but generally occurred in 30-40 age. The past, that the disease is a delayed epithelial moles, benign epithelial tumors, older skin, or changes in infectious diseases, but so far exact cause unknown. Some reports particular emphasis on family dominant inheritance. Clinical manifestations of the disease most common in the face, scalp, trunk, upper limbs, but can also occur in any part of the surface. Early damage to small and flat, the patch realm clear, smooth surface or slightly papilloma-shaped, yellowish brown. After the damage gradually increased, the surface was more tumor-like nipple, forming a layer of fat and thick callus. Pigmentation can be very significant. The color variation old great damage, a skin-color or short, even black. Hair follicle angle is an important feature of the suppository, and sometimes even small early rash In other words, it sees very clearly that the greater the damage to the surface from verrucous many small and flat papillary tumor-like damage from polymerization. Very thick crust formation damage, lightly surface crust Paper, a papillary tumor-like surface. Although the damage sustained grease-like surface, but there are many dry surface damage was verrucous. As superficial damage, it is like adhesive in the epidermis. The disease can be single, but usually more hair, the number of lesions can be many. Symptoms usually not consciously, sometimes itching. When the oil spill occurred in parts of friction or trauma sites, rash can be stimulating and epithelial tissue inflammation and irregular hyperplasia, also known as the stimulation of seborrheic keratosis. Damage occurred on the scalp, does not affect hair growth. The course usually slow, damage to the peripheral expansion, but also integration into large pieces. No self-healing tendencies. Although the report can be complicated with basal cell tumor, but seldom see, it is generally not considered to be precancerous lesions. Pathological changes from the pathology of the disease can be divided into three types, namely keratosis type, spine and adenoid hypertrophic layer type. The three often mixed exist. III have in common is the excessive keratosis, the spine of hypertrophy and tumor-like papillary hyperplasia. The features of the tumor lesions in the basal surface of the same level, the two ends connected with the normal epidermis. Spine - cell hypertrophy can be divided into two kinds of cells for the spine or squamous cell and the other for the basal-like cells, similar epidermal basal cells, such basal-like cells characteristic of this disease is. 3 changes also vary keratosis keratosis - are excessive and tumor-like papillary hyperplasia more notable, and spine of hypertrophy not obvious because of horny invagination of the results, so many false horny cyst formation. Spike hypertrophic layer keratosis are excessive and tumor-like papillary hyperplasia were lighter, although the foreseeable horny cyst, but more pronounced thickening of skin, the skin lesions are often thickened rough reticular formation, thickened epidermal basal cells in more than squamous cell. Adenoid-thin beam to strip from the majority of epidermal cells, such beam of the epidermis to the dermis by extension, and branches or cutting each other; bundle of cells by more than two-tier composed of basal cells. This type of keratocyst rare. Contained within the tumor cells with various types of pigment vary keratosis not obvious increase of pigment, the pigment-adenoid more spine while the number of hypertrophic layer mixed. If the disease combined with clinical and pathologic diagnosis is not difficult. But some early damage like flat wart; keratosis in the exposed parts of damage easy and senile keratosis (or solar keratosis of the disease) confused; pigment deep damage with nevus cells moles distinction. The inflammation or damage may stimulate similar basal cell or squamous cell carcinoma tumors and malignant melanoma, this time for biopsy or surgery required after pathological examination to identify. The disease generally does not require treatment, but only the beauty issue. If itching or inflammation, or diagnosis of a problem, or surgical resection. In addition, the available liquid nitrogen, carbon dioxide frozen treatment, but if not yet clear diagnosis, the best treatment for a biopsy first, so as to avoid misdiagnosis.

X-linked ichthyosis unusual performance

X-linked ichthyosis patients showed unusual rare. As the disease gene in the X chromosome, is found only in males, may infant after birth or morbidity. Lesions on the type and slightly different large and significant, a tawny or sewage black large, dry rough skin, often around the body, armpit, elbow Waterloo and other units can be involved, especially abdominal weight than the back. If facial involvement is limited to the ears before and facial aspect. Hair follicle keratosis not normally occur. Palmoplantar from normal skin, skin lesions did not increase with age and reduce, and sometimes it by drama. Female carriers of the disease, does not generally incidence, sometimes only found in the forearm and leg appeared thin. After flexibility and posterior corneal layer membrane turbidity can be a small point, and do not affect vision.

Lamellar ichthyosis clinical manifestations

Lamellar ichthyosis of autosomal recessive genetic, which is a whole body after birth extensive cotton rubbery membrane were tightly wrapped, multi-valgus cause eyelids and lips. A few days later the membrane off, a broad indemnity chronic skin flushing, there are gray or polygonal or rhomboid large, central fixation, the free edge. Often symmetry of the body to physically flexor side, elbow Waterloo, the Waterloo, armpit and genital and other units more distinctly. Palmoplantar hyperkeratosis, fingernails and excessive hair growth. After slow course, there could be life to adulthood erythroderma ease, but still exist. The disease is autosomal recessive inheritance.

The etiology of pathological ichthyosis

Generally considered with neuropsychiatric factors, endocrine disorders, immunological abnormalities, gastrointestinal disorders, etc., Fish disease causes can be summarized into several factors: genetic, mental nerve, chemical, endocrine, or outside factors were infected factors, and so on, we can see that the incidence of the disease to many factors, but we can not and what factors are related to any micro-site the circulatory disturbance, we study drugs, work hard to improve the microcirculation in this regard. Neural factors on the impact of emotional health. In our 10,000 cases on the diagnosis and treatment of cases, induced by the spirit of the reasons for the cases of 35%, and some have stimulated by the spirit, the spirit of some of the excessive tension, there is too much thinking suppressed.
Histopathological
Unusual-scale thinning disease manifested as skin, the stratum corneum mild to moderate thickening, particle layer to reduce or lack of hair follicles and sweat glands hole can be horny embolization, sebaceous reduction; of the hidden ichthyosis performance for Kok and granular layer thickening, highlighted a nail, a uniform distribution perivascular infiltration of lymphocytes, a slight decline in the number of sweat glands; congenital ichthyosis bullous disease manifested as red-keratosis excessive and spine of hypertrophy, particle layer containing coarse particles , the granular layer and the upper part of a spike layer mesh vacuolization, within the foreseeable blisters skin, the dermis shallow little inflammatory cell infiltration; lamellar ichthyosis showed moderate excessive keratosis, a focal part of keratosis insufficiency, particle layer thinning or slightly thickened, spine-moderate hypertrophy, a leather upper inflammatory cell infiltration; non-congenital ichthyosis bullous disease manifested as red-over-keratosis, accompanied by mild keratosis incomplete and spine of hypertrophy , dermal shallow lymphocyte infiltration.

Elderly canker disease which should be differentiated from?

(1) functional dyspepsia. This means there are no symptoms of ulcer dyspepsia or other organic diseases, such as chronic gastritis, duodenitis, or biliary disease. Accompanied with anxiety, insomnia, nervous tension, depression, may induce or aggravate mental factors, the anti-ulcer treatment effective medicine taking gastrointestinal motility drugs can significantly ease. The disease in young women is common in the elderly is not uncommon, should be aware of the differential. (2) of gastric cancer. Benign gastric ulcer and malignant ulcer (stomach cancer) is sometimes difficult to identify. Some ulcer in the early gastric cancer, and the morphological and clinical manifestations of benign gastric ulcer is similar, and even the anti-ulcer healing after treatment temporarily, to the early diagnosis difficult. Benign and malignant gastric ulcer main basis for the differential X-ray and endoscopic and pathologic examination, and therefore, for all elderly patients with gastric ulcer should visit gastroscopy, and in multiple ulcers from some marginal biopsy to determine the nature of ulcers. (3) chronic gastritis. The peptic ulcer disease may have similar symptoms easily confused with the. Analysis of gastric acid found lack of peptic ulcer can be ruled out, the higher the incidence of this disease, gastroscopy is the identification of the two most important methods. (4) chronic duodenitis. Has not yet sure whether the disease could be developed for the duodenal ulcer. Both clinical manifestations and is similar to X-ray findings, the diagnosis of duodenal mainly rely on endoscopic examination. (5) Gastroptosia. The disease can be accompanied by kidney, liver and other organs to sag, and a dyspeptic symptoms. All its aggravating symptoms in the upright, lying at ease. X-ray with barium meal examination of the X-ray findings of ulcer, gastric and small bending the lowest point in the iliac crest level below is the basis for the diagnosis of this disease. (6) chronic bile duct disease. Biliary motor dysfunction can cause seizures spastic right upper quadrant pain, the pain occurs after a meal in the application of basic drugs not eased. If asked in detail about history, the gallbladder with X-ray angiography, B-γ hepatobiliary scintillation camera, and other inspections, a clear diagnosis.

Purulent meningitis-related knowledge

Purulent meningitis is caused by a variety of bacterial meningitis suppurative inflammation. With fever, vomiting, headache, irritability, and with meningeal irritation and meningeal spinal fluid change as the main clinical features. The disease is common in babies infected by the disease. Lead to the disease pathogen species and age-related disease, several freshmen to Escherichia coli, Staphylococcus aureus encephalitis; infants to Streptococcus pneumoniae, Haemophilus influenzae encephalitis3-year-old children were more than Staphylococcus aureus lead to. Most of this rapid onset, severe illness, easily emerge Jingfeng, coma, a higher series of deaths and the incidence of sequelae. [Clinical] 1. E. coli meningitis: a predilection for birth within the last three babies, especially newborns and premature infants susceptible. Bacteria mainly from the mother's birth and infant intestinal Road, umbilical, suffering from abdominal baits are more disease, poor prognosis and mortality high. 2. Haemophilus influenzae meningitis:three months-3-year-old children, many of the autumn. Most of rapid onset, sudden high fever, vomiting, convulsions. Some onset gentler, prior apparent respiratory infection in a few days or 1-2 weeks after meningitis disease, even can see with rash. 3. Streptococcus pneumoniae is a higher incidence of meningitis after meningitis, more common in infants under 1 year of age, and often in the winter and spring seasons in the secondary respiratory inflammation, sepsis or traumatic brain injury, not obvious early stiff neck, the course can be deferred or repeated seizures. Can be complicated by subdural effusion or empyema, brain abscess, such as hydrocephalus, the general condition heavier. 4. Staphylococcus encephalitis: mainly caused by staphylococcus aureus, the incidence of all ages are more occurred in the summer. Often omphalitis, impetigo, cellulitis, and other precursor infection may be associated with pneumonia, lung abscess, liver abscess, suppurative osteomyelitis, and other lesions. Of course, there may be urticaria, scarlet fever rash and pustular lesions such as. Children of different ages have different features, clinical manifestations: 1. Neonates and infants under three months is not a very typical clinical manifestations. High temperature can be low, and even the temperature does not rise, often Antifeedant, vomit milk, lethargy, Ti panic, screams, convulsions and looking Jinghui, the former on existing full or uplift, meningeal irritation appeared late. 2.3, -2-year-old children with fever, vomiting, irritability, Yijiaore, convulsions, mental sluggishness, drowsiness or coma. Stiff neck, the former on existing Penglong, there meningeal irritation. 3,2 children over the age of symptoms and signs typical growing, with the exception of a headache, there are back pain, muscle pain and other joint complaint, meningeal irritation clear.

B meningitis in the early signal

B meningitis, transmitted mainly through mosquito bites. Disease-children under the age of 10, especially the 3-year-old to 6-year-old the highest incidence of children, young adults, the elderly nor rare disease. Common early symptoms are: A sudden fever. Patients with onset of intense, the temperature rose rapidly to more than 39 ℃, and persistent. 2, severe headache. Most patients with severe headache, irritability, crying more than the children. 3, jet-like vomiting. Because intracranial hypertension patients, there will be nothing to do with the diet of projectile vomiting. In the epidemic season, once found, these signals should be promptly taken to the hospital treatment, to prevent delay, otherwise, with the development of disease, the patient may appear lethargy and convulsions, coma, stiff neck, body spasm, and respiratory symptoms, such as the weakening of life-threatening .

Drug-induced meningitis alert

Generally believed that bacteria, viruses and lead to meningitis is the culprit. However, some drugs can also cause meningitis, medically known as the "drug-induced meningitis" or "aseptic meningitis." In recent years, clinicians found that some patients in the use of certain drugs will be similar to meningitis symptoms, such as fever, headache, stiff neck, lethargy, the sense of ambiguity, and even coma. Some patients showed a visible conjunctival hyperemia, nausea, vomiting, sweating, body pain and meningeal irritation symptoms of some patients neck pain, limb tendon reflexes symmetry hyperthyroidism, or convulsive spasm limbs, shortness of breath, depending on Fuzzy, that the proliferation of body rash. The peripheral blood and cerebrospinal fluid examination, can be found in erythrocyte sedimentation rate accelerated increase in cerebrospinal fluid interleukin such as abnormal changes, but found no special bacterial culture. The experts found that it is drug-induced meningitis. According to clinical statistics show that, ibuprofen, sulfamethoxazole, trimethoprim amine pyrimidine, caused the most reported drug-induced meningitis. In addition, the non-phenazopyridine, Tolmetin, immunoglobulin, cytarabine, isoniazid, penicillin, azathioprine, diclofenac sodium, pipemidate, also cause meningitis reported. Drug-induced meningitis mechanism is not yet known, but scholars tend to drug-induced allergic reaction, the reasons are: meningitis medication after clinical signs and symptoms of the short time interval, drug excited shorter latency, and severity of the increase, suspended after drug treatment without symptoms and signs quickly returned to normal. CSF after the drug stimulated immune complexes showed positive results, immunoglobulin synthesis rate increase, presumably because of drugs as a hapten combined with the protein to form a security antigen recognition by the immune system, thereby activating the brain sheath in the original autoantibodies in the immune response. Drug-induced meningitis occurs in adults and children is extremely rare, more women Most of cotrimoxazole such as meningitis caused by drug-induced occurred in almost all women, which may be related to the level of frequency of administration. Drug-induced meningitis diagnosis of a clear, we must immediately stop. Usually in the 12 ~ after treatment within 48 hours of their own rehabilitation. Only a small number of people needed antibiotics, hormones such as a symptomatic treatment. Patient prognosis is good, generally without sequelae.

Antibiotic treatment of the five principles of meningitis

Meningitis is a central nervous system infection, a higher mortality rate, the key to effective treatment is strict adherence to the five principles of antibiotics. 1, use fungicides. Meningeal infection in humans usually weak regional defense functions, the existence of the blood-brain barrier because of the lymphatic system and the reasons for the humoral and cellular immune dysfunction, and the absence of specific antibodies and complement, the application of antibacterial drugs therefore very poor results, only Application of fungicides can be a good way to remove bacteria in the cerebrospinal fluid. 2, use of drugs through the blood-brain barrier. Based on various antibacterial drugs through the blood-brain barrier into the cerebrospinal fluid concentrations, can be divided into four categories: general dose to be effective in the cerebrospinal fluid in the main concentration of chloramphenicol, sulfonamides, metronidazole, and isoniazid, rifampicin, ethambutol, and other anti-TB drugs; large intravenous dose of up to the main concentration of penicillin, ampicillin, cephalosporins, as well as ciprofloxacin, sparfloxacin oxygen Star; larger dose in the cerebrospinal fluid in only trace amount of the drug gentamicin, erythromycin, vancomycin; not measured in cerebrospinal fluid polymyxin how the drug, amphotericin B, Lin such as Adriamycin. 3, to avoid adverse reactions greater local administration. Including intrathecal administration and intraventricular administration. Only suffering from cryptococcal meningitis can be supplemented with amphotericin B ventricle or intrathecal administration. 4, the development of rational drug programme. Septic cerebrospinal fluid may be due to weakening of the antibacterial activity of antibiotics, in the development of treatment options, to consider using dose so that the drug concentration in cerebrospinal fluid to maintain a relatively high level. Intravenous route of administration in the main, intermittent or sustained. Of particular note was the improvement in the condition when the reduction should not arbitrarily, as it is infection control, reduce inflammation, blood-brain barrier permeability also will be reduced, if this reduction is difficult to maintain an effective antibiotic concentrations in cerebrospinal fluid, therefore need Application of the whole adequate antibiotics. 5, should achieve an effective drug treatment. Short-term antibiotic treatment high recurrence rate. To develop treatment varies for different pathogens: such as meningococcal meningitis caused treatment for 5 - 7 days, Haemophilus influenzae meningitis not less than 10 days, pneumococcal meningitis should be 14 days, and Gram-negative treatment of meningitis caused by at least 3 - 4 weeks. Indications for its withdrawal in cerebrospinal fluid cell count and biochemical indicators basically returned to normal, smear and culture negative bacteria. In short, bacterial meningitis antibiotic treatment must adhere to the "early, full, adequate, safe and effective" in order to improve the cure rate and lower mortality.