Simple leg varicose

Overview leg varicose common means lower limb superficial vein system Yu Qu, elongation and expansion. Primary deep venous insufficiency, Deep vein thrombosis and simple, such as varicose veins of lower limbs can cause varicose veins of lower limbs. Simple varices, said that to the primary varicose vein, the only means lesions in superficial venous leg. Most occurred in the great saphenous vein, with a few small saphenous vein or separately in small saphenous vein. Cause pathogenesis a lower extremity venous anatomy and the physiological characteristics into deep venous leg vein and two superficial vein. Deep vein in the muscle between the artery with the same name accompanied. Fascia superficial vein in shallow surface, sub-great saphenous vein and the small saphenous vein. Origin great saphenous vein in the medial dorsal vein network, the front foot, the medial leg up and Waterloo Park through the egg, into the femoral vein. Shares in the vein before a 5-7cm, 3 ~ 7 branches, and to five most common, The difference of abdominal superficial vein, shallow iliac vein and obturator vein, femoral vein and femoral medial vein. Starting small saphenous vein from the foot of the net lateral vein, along the lateral malleolus after Sural uplink, in the popliteal fossa through the deep fascia into the popliteal vein (Figure 2 -114). In deep and superficial vein, and between large and small saphenous vein between, there are many traffic branch vein mutual communication. Big leg deep vein of traffic support, mainly in sartorius muscle, the adductor muscle of the knee and 3; Small foot traffic to the leg vein and lateral malleolus traffic vein is most important. The foot traffic has three vein draining the lower leg 1 / 3 side of the blood directly into the posterior tibial vein. Lateral malleolus vein 431/490 traffic, drainage legs under 1 / 3 of blood outside perspective, direct access to Recife vein. Its valve insufficiency, and large and small saphenous vein and the incidence of venous stasis ulcer formation are closely related. Large and small saphenous vein between the traffic branch, located in the vicinity of the knee. Figure 2 -114 A lower extremity venous anatomy. Great saphenous vein and its Friends; B. Small saphenous vein in deep, superficial vein and transport support vein, the valve is there. Saphenous vein into the femoral vein near the small saphenous vein into the popliteal vein opening, and the deep vein branch vein, have both a strong presence valve. These one-way valve was open and maintain blood flow from distal to proximal or from shallow to deep flow. If the valve dysfunction occurred, the blood themselves there varices. Under normal circumstances, lower extremity venous return to the heart, rely on the heart beat generated by contraction, muscle contraction pump function and the chest when breathing, such as vacuum suction three combinations. Valve in the process of returning the blood to make it a one-way flow, without reflux occurred. 2, etiology and pathology of simple lower extremity varices causes of morbidity and venous insufficiency, vein weak and lasting intravenous pressure increased. Venous insufficiency of the major causes of venous valve defects. Defects and venous vein is weak support organizations body of a weak performance, and genetic factors. Lower extremity venous pressure caused lasting increase of the major reasons for this is a long standing and increased intra-abdominal pressure. Simple leg in the process of formation of varicose veins, venous vein with the intensity and the level of venous pressure, interaction plays a role. Venous and vein centrifuge far more, the lower intensity, the venous pressure is far more centrifugal were higher, therefore, varicose leg forward progress rapidly than the initial stage, and the expansion of tortuous superficial vein, smaller than in the leg thigh obvious. In simple varicose leg, the small saphenous vein has also been femoral popliteal and femoral vein valve protection, Xuezhu gravity will not be the direct impact only in saphenous vein varicose progress to a considerable extent, through the branches affected small saphenous vein, only in small saphenous vein regional distribution, showing varicose veins. Yu Qu leg vein, the expansion of the blood returning slowly, or even themselves occurred stasis and venous pressure increased. Nutrition vein occurred obstacles and degeneration, in particular vascular middle of muscle fiber atrophy and degeneration of elastic fibers. by connective tissue substitutes. Some vein cystic expansion was thinning, some positions because of connective tissue and thickening, which was vascular be nodular. Venous shrinking machine, and loss of function. Stagnant bloodstream and venous pressure and increased permeability of the capillary wall, intravascular fluid, protein, RBC and metabolites exudative to subcutaneous tissue, hyperplasia and fibers caused pigmentation. Local tissue hypoxia caused malnutrition, reducing resistance and easy with dermatitis, eczema, ulcers and infections. These pathological changes occurred in the Ministry of adequate boots of the skin, usually in the advanced stage of disease appeared.